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Minkyoung Kim

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3 published item(s)

preprint2026arXiv

Bilevel Graph Structure Learning, Revisited: Inner-Channel Origins of the Reported Gain

Bilevel graph structure learning is widely understood to improve graph neural networks by jointly optimizing model parameters and a learned graph structure, with the resulting performance gain attributed to the rewired adjacency. We find that this attribution may be overstated: training-dynamics effects in the inner loop, rather than the rewiring itself, capture a substantial share of the gain. To establish this, we introduce frozen-$φ$, a control that freezes the graph while retaining the inner-loop training schedule. This decomposes the bilevel gain into an inner channel of $T$-step training dynamics with implicit gradient regularization and a graph channel of the graph rewiring itself. On spatio-temporal flow forecasting the inner channel matches or exceeds the full bilevel pipeline, accounting for 78-101% of the gain; on node classification it accounts for 37-44% under a Bernoulli edge-level parameterization. We also verify that classical spectral diagnostics can dissociate from task gain. We propose frozen-$φ$ as a standardized diagnostic for bilevel graph structure learning, with graph distillation as a method-agnostic complement. A three-precondition framework further predicts the sign of the bilevel gain on all six benchmarks.

preprint2018arXiv

Causal Inference in Disease Spread across a Heterogeneous Social System

Diffusion processes are governed by external triggers and internal dynamics in complex systems. Timely and cost-effective control of infectious disease spread critically relies on uncovering the underlying diffusion mechanisms, which is challenging due to invisible causality between events and their time-evolving intensity. We infer causal relationships between infections and quantify the reflexivity of a meta-population, the level of feedback on event occurrences by its internal dynamics (likelihood of a regional outbreak triggered by previous cases). These are enabled by our new proposed model, the Latent Influence Point Process (LIPP) which models disease spread by incorporating macro-level internal dynamics of meta-populations based on human mobility. We analyse 15-year dengue cases in Queensland, Australia. From our causal inference, outbreaks are more likely driven by statewide global diffusion over time, leading to complex behavior of disease spread. In terms of reflexivity, precursory growth and symmetric decline in populous regions is attributed to slow but persistent feedback on preceding outbreaks via inter-group dynamics, while abrupt growth but sharp decline in peripheral areas is led by rapid but inconstant feedback via intra-group dynamics. Our proposed model reveals probabilistic causal relationships between discrete events based on intra- and inter-group dynamics and also covers direct and indirect diffusion processes (contact-based and vector-borne disease transmissions).

preprint2017arXiv

Universal Components of Real-world Diffusion Dynamics based on Point Processes

Bursts in human and natural activities are highly clustered in time, suggesting that these activities are influenced by previous events within the social or natural system. Bursty behavior in the real world conveys information of underlying diffusion processes, which have been the focus of diverse scientific communities from online social media to criminology and epidemiology. However, universal components of real-world diffusion dynamics that cut across disciplines remain unexplored. Here, we introduce a wide range of diffusion processes across disciplines and propose universal components of diffusion frameworks. We apply these components to diffusion-based studies of human disease spread, through a case study of the vector-borne disease dengue. The proposed universality of diffusion can motivate transdisciplinary research and provide a fundamental framework for diffusion models.